K ATP Channel Regulators

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K(ATP) channel regulators: balanced diets include carbohydrates, proteins, and fats.

In addition to the steep voltage-dependent K 1 currents that play a role in repolarization of the heart, two major K conductances control the resting potential. The classical strong inward rectifier (IK1) is constitutively active and acts to stabilize the resting potential. During depolarization, the strong rectification of IK1 significantly reduces conductance, permitting a long action-potenti...

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Effect of ATP-sensitive K+ channel regulators on cystic fibrosis transmembrane conductance regulator chloride currents

The cystic fibrosis transmembrane conductance regulator (CFTR) is a Cl- channel that is regulated by cAMP-dependent phosphorylation and by intracellular ATP. Intracellular ATP also regulates a class of K+ channels that have a distinct pharmacology: they are inhibited by sulfonylureas and activated by a novel class of drugs called K+ channel openers. In search of modulators of CFTR Cl- channels,...

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Effect of ATP-Dependent K+ Channel Openers and Blockers on Serum Concentration of Aldosterone in Rats

There are many reports for involvement of ATP-sensitive potassium channels in pancreatic, cardiac and vascular smooth muscle cells. This study examined the effect of single doses of K+ channel openers diazoxide, minoxidil and K+ channel blockers chlorpropamide, glibenclamide on serum concentration of aldosterone in male rats. Blood samples were obtained 60 minutes after drug treatment and serum...

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A novel mitochondrial K(ATP) channel assay.

RATIONALE The mitochondrial ATP sensitive potassium channel (mK(ATP)) is implicated in cardioprotection by ischemic preconditioning (IPC), but the molecular identity of the channel remains controversial. The validity of current methods to assay mK(ATP) activity is disputed. OBJECTIVE We sought to develop novel methods to assay mK(ATP) activity and its regulation. METHODS AND RESULTS Using a...

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Molecular Analysis of ATP-sensitive K Channel Gating and Implications for Channel Inhibition by ATP

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ژورنال

عنوان ژورنال: Circulation Research

سال: 2001

ISSN: 0009-7330,1524-4571

DOI: 10.1161/hh0901.091206